gov.noaa.nmfs.inport:17794 eng UTF8 dataset Access Database Northwest Fisheries Science Center 206-860-3200 2725 Montlake Boulevard East Seattle WA 98112 USA https://www.nwfsc.noaa.gov WWW:LINK-1.0-http--link Website Website for this organization information resourceProvider Northwest Fisheries Science Center 206-860-3200 2725 Montlake Boulevard East Seattle WA 98112 USA nmfs.nwfsc.metadata@noaa.gov https://www.nwfsc.noaa.gov WWW:LINK-1.0-http--link NWFSC Home Website listed for Northwest Fisheries Science Center information pointOfContact 2025-05-15T19:16:15 ISO 19115-2 Geographic Information - Metadata Part 2 Extensions for imagery and gridded data ISO 19115-2:2009(E) Oceanographic and physiological parameters relating to Heterosigma akashiwo in Puget Sound - The Ecophysiology and Toxicity of Heterosigma akashiwo in Puget Sound: A Living Laboratory Ecosystem Approach Oceanographic and physiological parameters relating to Heterosigma akashiwo in Puget Sound 2011-05-01 creation 2017-02-13 publication NOAA/NMFS/EDM 17794 https://www.fisheries.noaa.gov/inport/item/17794 WWW:LINK-1.0-http--link Full Metadata Record View the complete metadata record on InPort for more information about this dataset. information Adams, Nicolaus G 206-860-6787 206-860-3335 2725 Montlake Blvd East Seattle WA 98112-2097 Nicolaus.Adams@noaa.gov originator https://www.webapps.nwfsc.noaa.gov/apex/parrdata/inventory/datasets/dataset/4075 WWW:LINK-1.0-http--link Oceanographic and physiological parameters relating to Heterosigma akashiwo in Puget Sound NWFSC Dataset Information page. Contains oceanographic parameters such as temperature, salinity, density, fluorescence and nutrient concentrations. Also contains information on the toxic elements of Heterosigma akashiwo and physiological conditions related to observed toxic effects. download tableDigital Over one half of the worlds fish production for human consumption currently comes from aquaculture, while wild fisheries yields are either stable or declining. Recurring threats from the raphidophyte, Heterosigma akashiwo Hada (Sournia) have caused extensive damage ($2-6 million per episode) to wild and net-penned fish of Puget Sound, Washington, and are believed to be increasing in scope and magnitude in this region, and elsewhere in the world over the past two decades. The mechanism of H. akashiwo toxicity is not well understood. The toxic activity of H. akashiwo has been attributed to the production of reactive oxygen species, brevetoxin-like compound(s), excessive mucus, or hemolytic activity; however these mechanisms are not confirmed consistently in all fish-killing events or cultured strains. The difficulty of conducting research with active, toxin-producing field populations of H. akashiwo have resulted in conflicting findings from those obtained in lab culture studies, thereby limiting the ability of fish farmers to respond to these episodic blooms. Collaborators in this project are: Vera Trainer (NWFSC), William Cochlan (San Francisco State University), Charles Trick (University of Western Ontario), and Mark Wells (University of Maine). The overall goal of this project is to identify the primary toxic element and the specific environmental factors that stimulate fish-killing H. akashiwo blooms, and thereby provide managers with the fundamental tools needed to help reduce the frequency and toxic magnitude of these harmful algal events. Studies to date have provided incomplete and conflicting observations on the mode of toxicity and the environmental stimulation of toxification. We propose a three-pronged approach to study the environmental controls of H. akashiwo growth and toxin production; laboratory culture experiments, field observations, and bottle and mesocosm manipulation experiments.The project objectives are to: 1. identify the element(s) of toxic activity (inorganic, organic, or synergistic) associated with blooms of H. akashiwo and the various cellular morphologies of this alga, 2. determine the environmental parameters that stimulate the growth success and expression of cell toxicity in the H. akashiwo populations of Puget Sound. Because previous studies have used H. akashiwo cultures with little or no toxic activity, our approach is to use a living laboratory to study H. akashiwo bloom ecology and toxicity using natural assemblages. Using a mobile lab at field sites where H. akashiwo cells are regularly found will enable us to fully characterize the toxic element(s) responsible for fish mortality, and the environmental factors influencing toxicity. Findings from annual field studies in June and two rapid response deployments during major bloom events will be confirmed using laboratory studies with fresh ( 6 mo. old) isolates. The expected results are: 1. determination of the key elements of toxicity of H. akashiwo, 2. characterization of the environmental variables that influence either the induction or depression of elements of toxic activity in H. akashiwo, 3. characterization of environmentally-induced metabolites corresponding to condition of toxin production (metabolomics) and 4. design of a strategy for realistic mitigation of H. akashiwo activities in Puget Sound, Washington. This is a stand-alone project funded for 3 years through the NOAA/NSF ECOHAB program. Contains oceanographic parameters such as temperature, salinity, density, fluorescence and nutrient concentrations. Also contains information on the toxic elements of Heterosigma akashiwo and physiological conditions related to observed toxic effects. Raw/field data, Monitoring or sensor data, Time series (of types 1-3), Laboratory data completed Adams, Nicolaus G 206-860-6787 206-860-3335 2725 Montlake Blvd East Seattle WA 98112-2097 Nicolaus.Adams@noaa.gov pointOfContact Adams, Nicolaus G 206-860-6787 206-860-3335 2725 Montlake Blvd East Seattle WA 98112-2097 Nicolaus.Adams@noaa.gov custodian marine toxin theme ECOREGION East Sound, Orcas Island Ecoregions Friday Harbor Laboratories Montlake NWFSC Montlake Puget Trough/Georgia Basin US Waters of Puget Sound/Georgia Basin place In-Situ Instrument - In-Situ Instrument (not specified) Sonde and Probe - Conductivity Temperature and Depth instrument Animal and Plant Collection Device Animal and Plant Collection Device - Plankton Collection Device - Plankton Net Water Sampler Bottle - Surface Sample Bottle platform DOC/NOAA/NMFS/NWFSC > Northwest Fisheries Science Center, National Marine Fisheries Service, NOAA. U.S. Department of Commerce dataCentre Global Change Master Directory (GCMD) Data Center Keywords 2017-04-24 publication 8.5 Earth Science Data and Information System, Earth Science Projects Division, Goddard Space Flight Center (GSFC), National Aeronautics and Space Administration (NASA) https://forum.earthdata.nasa.gov/app.php/tag/GCMD%2BKeywords GCMD Keyword Forum Page Global Change Master Directory (GCMD). 2025. GCMD Keywords, Version 22. Greenbelt, MD: Earth Science Data and Information System, Earth Science Projects Division, Goddard Space Flight Center (GSFC), National Aeronautics and Space Administration (NASA). URL (GCMD Keyword Forum Page): https://forum.earthdata.nasa.gov/app.php/tag/GCMD+Keywords information custodian Migrated from Environmental Conservation Division project InPort otherRestrictions Cite As: Northwest Fisheries Science Center, [Date of Access]: Oceanographic and physiological parameters relating to Heterosigma akashiwo in Puget Sound - The Ecophysiology and Toxicity of Heterosigma akashiwo in Puget Sound: A Living Laboratory Ecosystem Approach [Data Date Range], https://www.fisheries.noaa.gov/inport/item/17794. NOAA provides no warranty, nor accepts any liability occurring from any incomplete, incorrect, or misleading data, or from any incorrect, incomplete, or misleading use of the data. It is the responsibility of the user to determine whether or not the data is suitable for the intended purpose. otherRestrictions Access Constraints: NA unclassified NOAA Data Management Plan (DMP) NOAA/NMFS/EDM 17794 https://www.fisheries.noaa.gov/inportserve/waf/noaa/nmfs/nwfsc/dmp/pdf/17794.pdf WWW:LINK-1.0-http--link NOAA Data Management Plan (DMP) NOAA Data Management Plan for this record on InPort. information crossReference eng; US oceans Access Database -122.3062 -122.2962 47.6449 47.6549 NWFSC Montlake 2010-10-01 2013-09-30 -122.8963 -122.8863 48.6644 48.6744 East Sound, Orcas Island 2010-10-01 2013-09-30 -123.0144 -123.0044 48.5458 48.5558 Friday Harbor Laboratories 2010-10-01 2013-09-30 -125.3893 -119.9604 47.0437 50.5101 US Waters of Puget Sound/Georgia Basin 2010-10-01 2013-09-30 Subject to Public Access to Research Results (PARR): Yes Data: Determine the toxin produced by Heterosigma to allow tracking of blooms threatening fish farms. Data: Determine the environmental factors promoting toxicity in Heterosigma to allow forecasting of blooms. Peer Reviewed Publication: Research paper in special issue on HABs in Stratified Systems. false eng false Spreadsheet NOAA/NSF ECOHAB Heterosigma Discreete Data 2018-02-27 publication NOAA/NSF ECOHAB Heterosigma CTD 2018-02-27 publication Northwest Fisheries Science Center 206-860-3200 2725 Montlake Boulevard East Seattle WA 98112 USA nmfs.nwfsc.metadata@noaa.gov https://www.nwfsc.noaa.gov WWW:LINK-1.0-http--link NWFSC Home Website listed for Northwest Fisheries Science Center information distributor https://www.webapps.nwfsc.noaa.gov/apex/parr/noaansf_ecohab_heterosigma_discreete_data/data/page/ WWW:LINK-1.0-http--link NOAA/NSF ECOHAB Heterosigma Discreete Data (RESTfu Over one half of the worlds fish production for human consumption currently comes from aquaculture, while wild fisheries yields are either stable or declining. Recurring threats from the raphidophyte, Heterosigma akashiwo Hada (Sournia) have caused extensive damage ($2-6 million per episode) to wild and net-penned fish of Puget Sound, Washington, and are believed to be increasing in scope and magnitude in this region, and elsewhere in the world over the past two decades. The mechanism of H. akashiwo toxicity is not well understood. The toxic activity of H. akashiwo has been attributed to the production of reactive oxygen species, brevetoxin-like compound(s), excessive mucus, or hemolytic activity; however these mechanisms are not confirmed consistently in all fish-killing events or cultured strains. The difficulty of conducting research with active, toxin-producing field populations of H. akashiwo have resulted in conflicting findings from those obtained in lab culture studies, thereby limiting the ability of fish farmers to respond to these episodic blooms. Collaborators in this project are: Vera Trainer (NWFSC), William Cochlan (San Francisco State University), Charles Trick (University of Western Ontario), and Mark Wells (University of Maine). The overall goal of this project is to identify the primary toxic element and the specific environmental factors that stimulate fish-killing H. akashiwo blooms, and thereby provide managers with the fundamental tools needed to help reduce the frequency and toxic magnitude of these harmful algal events. Studies to date have provided incomplete and conflicting observations on the mode of toxicity and the environmental stimulation of toxification. We propose a three-pronged approach to study the environmental controls of H. akashiwo growth and toxin production; laboratory culture experiments, field observations, and bottle and mesocosm manipulation experiments.The project objectives are to: 1. identify the element(s) of toxic activity (inorganic, organic, or synergistic) associated with blooms of H. akashiwo and the various cellular morphologies of this alga, 2. determine the environmental parameters that stimulate the growth success and expression of cell toxicity in the H. akashiwo populations of Puget Sound. Because previous studies have used H. akashiwo cultures with little or no toxic activity, our approach is to use a living laboratory to study H. akashiwo bloom ecology and toxicity using natural assemblages. Using a mobile lab at field sites where H. akashiwo cells are regularly found will enable us to fully characterize the toxic element(s) responsible for fish mortality, and the environmental factors influencing toxicity. Findings from annual field studies in June and two rapid response deployments during major bloom events will be confirmed using laboratory studies with fresh ( 6 mo. old) isolates. The expected results are: 1. determination of the key elements of toxicity of H. akashiwo, 2. characterization of the environmental variables that influence either the induction or depression of elements of toxic activity in H. akashiwo, 3. characterization of environmentally-induced metabolites corresponding to condition of toxin production (metabolomics) and 4. design of a strategy for realistic mitigation of H. akashiwo activities in Puget Sound, Washington. This is a stand-alone project funded for 3 years through the NOAA/NSF ECOHAB program. download https://www.webapps.nwfsc.noaa.gov/apex/parrdata/inventory/tables/table/noaansf_ecohab_heterosigma_ctd WWW:LINK-1.0-http--link NOAA/NSF ECOHAB Heterosigma CTD Over one half of the worlds fish production for human consumption currently comes from aquaculture, while wild fisheries yields are either stable or declining. Recurring threats from the raphidophyte, Heterosigma akashiwo Hada (Sournia) have caused extensive damage ($2-6 million per episode) to wild and net-penned fish of Puget Sound, Washington, and are believed to be increasing in scope and magnitude in this region, and elsewhere in the world over the past two decades. The mechanism of H. akashiwo toxicity is not well understood. The toxic activity of H. akashiwo has been attributed to the production of reactive oxygen species, brevetoxin-like compound(s), excessive mucus, or hemolytic activity; however these mechanisms are not confirmed consistently in all fish-killing events or cultured strains. The difficulty of conducting research with active, toxin-producing field populations of H. akashiwo have resulted in conflicting findings from those obtained in lab culture studies, thereby limiting the ability of fish farmers to respond to these episodic blooms. Collaborators in this project are: Vera Trainer (NWFSC), William Cochlan (San Francisco State University), Charles Trick (University of Western Ontario), and Mark Wells (University of Maine). The overall goal of this project is to identify the primary toxic element and the specific environmental factors that stimulate fish-killing H. akashiwo blooms, and thereby provide managers with the fundamental tools needed to help reduce the frequency and toxic magnitude of these harmful algal events. Studies to date have provided incomplete and conflicting observations on the mode of toxicity and the environmental stimulation of toxification. We propose a three-pronged approach to study the environmental controls of H. akashiwo growth and toxin production; laboratory culture experiments, field observations, and bottle and mesocosm manipulation experiments.The project objectives are to: 1. identify the element(s) of toxic activity (inorganic, organic, or synergistic) associated with blooms of H. akashiwo and the various cellular morphologies of this alga, 2. determine the environmental parameters that stimulate the growth success and expression of cell toxicity in the H. akashiwo populations of Puget Sound. Because previous studies have used H. akashiwo cultures with little or no toxic activity, our approach is to use a living laboratory to study H. akashiwo bloom ecology and toxicity using natural assemblages. Using a mobile lab at field sites where H. akashiwo cells are regularly found will enable us to fully characterize the toxic element(s) responsible for fish mortality, and the environmental factors influencing toxicity. Findings from annual field studies in June and two rapid response deployments during major bloom events will be confirmed using laboratory studies with fresh ( 6 mo. old) isolates. The expected results are: 1. determination of the key elements of toxicity of H. akashiwo, 2. characterization of the environmental variables that influence either the induction or depression of elements of toxic activity in H. akashiwo, 3. characterization of environmentally-induced metabolites corresponding to condition of toxin production (metabolomics) and 4. design of a strategy for realistic mitigation of H. akashiwo activities in Puget Sound, Washington. This is a stand-alone project funded for 3 years through the NOAA/NSF ECOHAB program. download https://www.webapps.nwfsc.noaa.gov/apex/parr/noaansf_ecohab_heterosigma_ctd/data/page/ WWW:LINK-1.0-http--link NOAA/NSF ECOHAB Heterosigma CTD (RESTful) Over one half of the worlds fish production for human consumption currently comes from aquaculture, while wild fisheries yields are either stable or declining. Recurring threats from the raphidophyte, Heterosigma akashiwo Hada (Sournia) have caused extensive damage ($2-6 million per episode) to wild and net-penned fish of Puget Sound, Washington, and are believed to be increasing in scope and magnitude in this region, and elsewhere in the world over the past two decades. The mechanism of H. akashiwo toxicity is not well understood. The toxic activity of H. akashiwo has been attributed to the production of reactive oxygen species, brevetoxin-like compound(s), excessive mucus, or hemolytic activity; however these mechanisms are not confirmed consistently in all fish-killing events or cultured strains. The difficulty of conducting research with active, toxin-producing field populations of H. akashiwo have resulted in conflicting findings from those obtained in lab culture studies, thereby limiting the ability of fish farmers to respond to these episodic blooms. Collaborators in this project are: Vera Trainer (NWFSC), William Cochlan (San Francisco State University), Charles Trick (University of Western Ontario), and Mark Wells (University of Maine). The overall goal of this project is to identify the primary toxic element and the specific environmental factors that stimulate fish-killing H. akashiwo blooms, and thereby provide managers with the fundamental tools needed to help reduce the frequency and toxic magnitude of these harmful algal events. Studies to date have provided incomplete and conflicting observations on the mode of toxicity and the environmental stimulation of toxification. We propose a three-pronged approach to study the environmental controls of H. akashiwo growth and toxin production; laboratory culture experiments, field observations, and bottle and mesocosm manipulation experiments.The project objectives are to: 1. identify the element(s) of toxic activity (inorganic, organic, or synergistic) associated with blooms of H. akashiwo and the various cellular morphologies of this alga, 2. determine the environmental parameters that stimulate the growth success and expression of cell toxicity in the H. akashiwo populations of Puget Sound. Because previous studies have used H. akashiwo cultures with little or no toxic activity, our approach is to use a living laboratory to study H. akashiwo bloom ecology and toxicity using natural assemblages. Using a mobile lab at field sites where H. akashiwo cells are regularly found will enable us to fully characterize the toxic element(s) responsible for fish mortality, and the environmental factors influencing toxicity. Findings from annual field studies in June and two rapid response deployments during major bloom events will be confirmed using laboratory studies with fresh ( 6 mo. old) isolates. The expected results are: 1. determination of the key elements of toxicity of H. akashiwo, 2. characterization of the environmental variables that influence either the induction or depression of elements of toxic activity in H. akashiwo, 3. characterization of environmentally-induced metabolites corresponding to condition of toxin production (metabolomics) and 4. design of a strategy for realistic mitigation of H. akashiwo activities in Puget Sound, Washington. This is a stand-alone project funded for 3 years through the NOAA/NSF ECOHAB program. download https://www.webapps.nwfsc.noaa.gov/apex/parrdata/inventory/tables/table/noaansf_ecohab_heterosigma_discreete_data WWW:LINK-1.0-http--link NOAA/NSF ECOHAB Heterosigma Discreete Data Over one half of the worlds fish production for human consumption currently comes from aquaculture, while wild fisheries yields are either stable or declining. Recurring threats from the raphidophyte, Heterosigma akashiwo Hada (Sournia) have caused extensive damage ($2-6 million per episode) to wild and net-penned fish of Puget Sound, Washington, and are believed to be increasing in scope and magnitude in this region, and elsewhere in the world over the past two decades. The mechanism of H. akashiwo toxicity is not well understood. The toxic activity of H. akashiwo has been attributed to the production of reactive oxygen species, brevetoxin-like compound(s), excessive mucus, or hemolytic activity; however these mechanisms are not confirmed consistently in all fish-killing events or cultured strains. The difficulty of conducting research with active, toxin-producing field populations of H. akashiwo have resulted in conflicting findings from those obtained in lab culture studies, thereby limiting the ability of fish farmers to respond to these episodic blooms. Collaborators in this project are: Vera Trainer (NWFSC), William Cochlan (San Francisco State University), Charles Trick (University of Western Ontario), and Mark Wells (University of Maine). The overall goal of this project is to identify the primary toxic element and the specific environmental factors that stimulate fish-killing H. akashiwo blooms, and thereby provide managers with the fundamental tools needed to help reduce the frequency and toxic magnitude of these harmful algal events. Studies to date have provided incomplete and conflicting observations on the mode of toxicity and the environmental stimulation of toxification. We propose a three-pronged approach to study the environmental controls of H. akashiwo growth and toxin production; laboratory culture experiments, field observations, and bottle and mesocosm manipulation experiments.The project objectives are to: 1. identify the element(s) of toxic activity (inorganic, organic, or synergistic) associated with blooms of H. akashiwo and the various cellular morphologies of this alga, 2. determine the environmental parameters that stimulate the growth success and expression of cell toxicity in the H. akashiwo populations of Puget Sound. Because previous studies have used H. akashiwo cultures with little or no toxic activity, our approach is to use a living laboratory to study H. akashiwo bloom ecology and toxicity using natural assemblages. Using a mobile lab at field sites where H. akashiwo cells are regularly found will enable us to fully characterize the toxic element(s) responsible for fish mortality, and the environmental factors influencing toxicity. Findings from annual field studies in June and two rapid response deployments during major bloom events will be confirmed using laboratory studies with fresh ( 6 mo. old) isolates. The expected results are: 1. determination of the key elements of toxicity of H. akashiwo, 2. characterization of the environmental variables that influence either the induction or depression of elements of toxic activity in H. akashiwo, 3. characterization of environmentally-induced metabolites corresponding to condition of toxin production (metabolomics) and 4. design of a strategy for realistic mitigation of H. akashiwo activities in Puget Sound, Washington. This is a stand-alone project funded for 3 years through the NOAA/NSF ECOHAB program. download dataset Accuracy High NA