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    • Data set fro Inhibition of Human ABC efflux transporters p-gp and BCRP by BDE-47 hydroxylated metabolite 6-OH-BDE-47

    Metadata Updated: November 12, 2020

    In vitro data from cell-based assays on the inhibition of BDE and its hydroxylated metabolite on ABC transporters.

    This dataset is associated with the following publication: Marchitti, S., C. Mazur, C. Dillingham, S. Rawat, A. Sharma, J. Zastre, and J. Kenneke. Inhibition of the Human ABC Efflux Transporters P-gp and BCRP by the BDE-47 Hydroxylated Metabolite 6-OH-BDE-47: Considerations for Human Exposure. TOXICOLOGICAL SCIENCES. Society of Toxicology, 155(1): 270-282, (2017).

    Access & Use Information

    Public: This dataset is intended for public access and use. License: See this page for license information.

    Downloads & Resources

    References

    https://doi.org/10.1093/toxsci/kfw209

    Dates

    Metadata Created Date November 12, 2020
    Metadata Updated Date November 12, 2020

    Metadata Source

    Harvested from EPA ScienceHub

    Other Data Resources

    Additional Metadata

    Resource Type Dataset
    Metadata Created Date November 12, 2020
    Metadata Updated Date November 12, 2020
    Publisher U.S. EPA Office of Research and Development (ORD)
    Maintainer
    Identifier https://doi.org/10.23719/1407655
    Data Last Modified 2016-09-13
    Public Access Level public
    Bureau Code 020:00
    Schema Version https://project-open-data.cio.gov/v1.1/schema
    Harvest Object Id 17582483-5c2e-4111-a1d4-299ed4830c9a
    Harvest Source Id 04b59eaf-ae53-4066-93db-80f2ed0df446
    Harvest Source Title EPA ScienceHub
    License https://pasteur.epa.gov/license/sciencehub-license.html
    Program Code 020:095
    Publisher Hierarchy U.S. Government > U.S. Environmental Protection Agency > U.S. EPA Office of Research and Development (ORD)
    Related Documents https://doi.org/10.1093/toxsci/kfw209
    Source Datajson Identifier True
    Source Hash d9fb49db2abb0300438bb4d3efb32f4c230cf72e
    Source Schema Version 1.1

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